IABDM Biohacking Conference Session 5: Intracellular Heavy Metal Chelation, Mercury Toxicity & the OSR Compound

Course Title: IABDM Biohacking Conference Session 5: Intracellular Heavy Metal Chelation, Mercury Toxicity & the OSR Compound

Date Recorded: 3/28/2026

Date Reviewed:  

Date of Expiration: 

Location: Online

Method: Self Directed

#CE HOURS: 1

Speaker(s) & Bio: 

Dr. David Kennedy, DDS

Dr. David Kennedy is a Past President of the International Academy of Oral Medicine and Toxicology.  He earned a degree in Comparative Biochemistry and Physiology from the University of Kansas and a Doctorate of Dental Surgery from the University of Missouri.

in 1973, Dr. Kennedy started a private practice in San Diego.  He also established the Chicano Children’t Dental Health Center.  A decade later, he founded the Preventive Dental Health Association.

Dr. Kennedy continues to lecture on the safety of dental materials in the body.

Course Description:

This lecture provides an in-depth examination of mercury as a systemic toxin, with a focus on the biochemical mechanisms by which heavy metals — particularly mercury — impair mitochondrial function through oxidative stress and electron disruption at the cellular level. Participants will explore the critical distinction between true intracellular chelation and extracellular migration strategies, and why this difference matters clinically. The presentation reviews the development and safety profile of OSR (oxidative stress relief), a fat-soluble, two-armed glutathione-derived compound developed by Dr. Boyd Haley, which functions as a selective intracellular chelator capable of neutralizing heavy metals, free iron, and free copper while demonstrating antioxidant activity exceeding 200,000 ORAC units. Clinical trial data and animal study findings are reviewed, along with the dose-dependent relationship between amalgam mercury vapor exposure and measurable neurocognitive and physiological impairment in dental personnel and pediatric populations. The presentation addresses the role of genetic susceptibility variants (APO4, CPOX4, MTHFR), the mercury-selenium-Alzheimer’s connection, mercury-associated demyelination and cardiomyopathy, andthe ethical and regulatory failures surrounding amalgam use and controlled studies in vulnerable populations. Occupational exposure risks for dental professionals — including hygienists, assistants, and dentists — are discussed in the context of OSHA regulations and workplace safety obligations.

Course Objectives:

Upon completion of this session, participants will be able to:

1. Distinguish between true chelation and migration of heavy metals, and explain why conventional water-soluble chelators (DMPS, DMSA) are limited in their ability to address intracellular metal burden.
2. Describe the mechanism of action of the OSR (oxidative stress relief) compound developed by Dr. Boyd Haley, including its selectivity for toxic versus essential metals.
3. Explain how mercury causes neurological damage, including demyelination, and identify clinical conditions associated with chronic mercury intoxication.
4. Identify the documented clinical outcomes — including reduced fatigue, tremor, and urinary mercury — observed in human trials of the OSR compound.
5. Articulate the relationship between dental amalgam, occupational mercury exposure in dental settings, and systemic health consequences for both patients and dental professionals.

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